Non-medullary thyroid carcinoma [12 genes]

The vast majority of non-medullary thyroid carcinomas develop sporadically. However, a small percentage of cases present familial aggregation. The association of non-medullary thyroid carcinoma with different entities of genetic aetiology has been confirmed. If a predisposition syndrome to non-medullary thyroid cancer is suspected, the following gene panel is recommended.

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Non-medullary thyroid carcinoma

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Technique: NGS capture panel

Turnaround time (TAT): 15 days

Ref. S-202211935

Updated (dd/mm/yy): 14/10/2025

Non-medullary thyroid carcinoma: View panel
  • AKT1
  • APC
  • DICER1
  • HABP2
  • KLLN
  • PIK3CA
  • PTEN
  • SDHB
  • SDHC
  • SDHD
  • SEC23B
  • PRKAR1A

Priority Genes : Genes where there is sufficient evidence (clinical and functional) to consider them associated with the disease; they are included in the clinical practice guidelines.
Secondary Genes: Genes related to the disease, but with a lower level of evidence or that constitute sporadic cases.
* Candidate Genes: Not enough evidence in humans, but potentially associated with the disease.

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